Traumatic brain injury (TBI) enhances pro-inflammatory responses, neuronal loss and long-term behavioral deficits. Caveolins (Cavs) are regulators of neuronal and glial survival signaling.
Previously we showed that astrocyte and microglial activation is increased in Cav-1 knock-out (KO) mice…
Traumatic brain injury enhances neuroinflammation and lesion volume in caveolin deficient mice
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